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Hydrocortisone aceponate (Hydrocortisone 17-propionate 21-acetate) is a potent topical glucocorticoid supplied as a research-grade analytical standard in solid powder and solution forms. It is used for studies of glucocorticoid receptor activity and for research into inflammatory dermatoses. Molecular formula C26H36O7; molecular weight 460.56; CAS 74050-20-7.
Potent topical glucocorticoid for dermatoses research.
Provided as powder (solid) and as 10 mM solution in DMSO.
Intended for research and analytical applications (research use only).
This product is provided as delivered and specified by the issuing Pharmacopoeia. All information provided in support of this product including SDS and any product information leaflets have been developed and issued under the Authority of the Issuing Pharmacopoeia. for further information and support please go to the website of the issuing Pharmacopoeia.
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Lignoceric acid (tetracosanoic acid) is a 24-carbon saturated (24:0) long-chain fatty acid supplied as a solid for laboratory use. It is used as a reagent and reference standard in lipid research and analytical chemistry.
24-carbon saturated fatty acid (C24:0).
Molecular weight 368.64 g/mol; formula C24H48O2.
High purity, listed as 98.0%.
Available in small pack sizes suitable for analytical work, including 100 mg.
Solid, white to off-white appearance.
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A product of the action of 15- and 5-LO on DHA; dampens excessive neutrophil trafficking to sites of inflammation; reduces zymosan-stimulated PMN infiltration by 70% at 10 pg per mouse; reduces PAF-stimulated leukocyte adherence and emigration at 1 nM; stimulates nitric oxide production decreases leukocyte-endothelial interactions; reduces leukocyte and PMN infiltration, decreases production of pro-inflammatory cytokines, and promotes phagocyte-mediated bacterial clearance in a mouse model of sepsis
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A saturated fatty acid methyl ester; concentration in cells is modulated by methanol; inhibits phagocytosis and decreases cell viability in isolated Kupffer cells; decreases secretion of IL-10, TNF-α, NO, and PGE2 in LPS-treated cells
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